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The elderly, in particular, are susceptible to poor outcomes after burns, due primarily to a scarcity of knowledge on the biomolecular effects of trauma in this population. Objectives: Severe burn injuries are characterized by alterations in mitochondrial dynamics which coincide with the ‘ebb’ and ‘flow’ phase of metabolic progression following trauma. 1Sunnybrook Health Sciences Centre, Toronto, Canada 2Ross Tilley Burn Centre, Toronto, Canada and 3University of Toronto, Toronto, Canada METFORMIN CORRECTS HEPATIC MITOCHONDRIAL ABNORMALITIES IN A MURINE MODEL OF THERMAL TRAUMA Christopher Auger 1,2, Thibacg Sivayoganathan 1,2,3, Abdikarim Abdullahi 1,2,3, Alexandra Parousis 1,2 and Marc G.
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Still, there remains potential to monitor the immunometabolic state over time and intervene with immune- and metabolic-targeted therapies as novel therapeutic approaches in sepsis. However, the factors that drive mitochondrial changes and the role by which mitochondrial energy metabolism regulates immune cell functions during the host response to infection remain unclear. Our data suggests that decreased mitochondrial content leads to a reduction in mitochondrial respiration.
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Changes in mitochondrial energy metabolism are evident within leukocyte subsets in sepsis, suggesting that metabolic processes could be a novel therapeutic target. Within individual patients, a differential balance in these immune components can produce either a hyperinflammatory or an immunoparalytic phenotype. The acute phase of sepsis is driven by a strong immune response with both pro- and anti-inflammatory components. 1Children's Hospital of Philadelphia, Philadelphia, PA 2Center for Mitochondrial and Epigenomic Medicine, Philadelphia, PA and 3University of Pennsylvania Perelman School of Medicine, Philadelphia, PA
#Express burn plus 4.68 serial pdf
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